Bimodal effect on pancreatic β-cells of secretory products from normal or insulin-resistant human skeletal muscle

Karim Bouzakri; Peter Plomgaard; Thierry Berney; Marc Y Donath; Bente Klarlund Pedersen; Philippe A Halban

doi:10.2337/db10-1178

Bimodal effect on pancreatic β-cells of secretory products from normal or insulin-resistant human skeletal muscle

Karim Bouzakri, Peter Plomgaard, Thierry Berney, Marc Y Donath, Bente Klarlund Pedersen, Philippe A Halban

Publikation: Bidrag til tidsskrift › Tidsskriftartikel › Forskning › peer review

103 Citationer (Scopus)

Abstract

Type 2 diabetes is characterized by insulin resistance with a relative deficiency in insulin secretion. This study explored the potential communication between insulin-resistant human skeletal muscle and primary (human and rat) ß-cells.

Originalsprog	Engelsk
Tidsskrift	Diabetes
Vol/bind	60
Udgave nummer	4
Sider (fra-til)	1111-21
Antal sider	11
ISSN	0012-1797
DOI	https://doi.org/10.2337/db10-1178
Status	Udgivet - 2011

Adgang til dokumentet

10.2337/db10-1178

Citationsformater

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title = "Bimodal effect on pancreatic β-cells of secretory products from normal or insulin-resistant human skeletal muscle",

abstract = "Type 2 diabetes is characterized by insulin resistance with a relative deficiency in insulin secretion. This study explored the potential communication between insulin-resistant human skeletal muscle and primary (human and rat) {\ss}-cells.",

author = "Karim Bouzakri and Peter Plomgaard and Thierry Berney and Donath, {Marc Y} and Pedersen, {Bente Klarlund} and Halban, {Philippe A}",

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AU - Bouzakri, Karim

AU - Plomgaard, Peter

AU - Berney, Thierry

AU - Donath, Marc Y

AU - Pedersen, Bente Klarlund

AU - Halban, Philippe A

PY - 2011

Y1 - 2011

N2 - Type 2 diabetes is characterized by insulin resistance with a relative deficiency in insulin secretion. This study explored the potential communication between insulin-resistant human skeletal muscle and primary (human and rat) ß-cells.

AB - Type 2 diabetes is characterized by insulin resistance with a relative deficiency in insulin secretion. This study explored the potential communication between insulin-resistant human skeletal muscle and primary (human and rat) ß-cells.

U2 - 10.2337/db10-1178

DO - 10.2337/db10-1178

M3 - Journal article

SN - 0012-1797

VL - 60

SP - 1111

EP - 1121

JO - Diabetes

JF - Diabetes

IS - 4

ER -