Physical Functioning Report and Pulmonary Function Deficit in Childhood and Adolescent Acute Lymphoblastic Leukemia Survivors: A National Cross-Sectional ALL-STAR Lungs Study

Sonja Izquierdo Riis Meyer*, Mette Tiedemann Skipper, Birgitte Klug Albertsen, Ruta Tuckuviene, Peder Skov Wehner, Thomas Leth Frandsen, Kjeld Schmiegelow, Liv Andrés-Jensen, Kim Gjerum Nielsen, Sune Leisgaard Mørck Rubak

*Corresponding author af dette arbejde

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Abstract

Background
Childhood acute lymphoblastic leukemia (ALL) survivors face an increased risk of pulmonary function deficit (PFD), which may affect physical functioning. We aimed to evaluate parent proxy- and self-reported physical functioning and the association with PFD and abnormal bronchodilator response ( + BDR) among survivors versus controls.

Methods
This cross-sectional study (February 2019–May 2024) included N = 184 ALL survivors (aged 5–17.9 years) from a national NOPHO ALL2008 protocol-treated cohort (eligible, N = 295) ( ≥ 1 year after treatment cessation) and 207 controls. Physical functioning was assessed using the Paediatric Quality of Life Inventory (PedsQL) questionnaire. Pulmonary function tests (PFTs) identified PFD (abnormal PFT) and +BDR according to ERS/ATS criteria. Ordinal logistic regression and t-test compared groups.

Results
ALL survivor parents proxy-reported significantly poorer physical functioning than controls (mean difference [MD] −13.3 95% CI [−16.3; −10.3] to MD −10.3 [−17.2; −3.3], p < 0.001 to p = 0.004), but clinically significant impairments were restricted to parent proxy-reports from survivors treated with high-risk (HR) chemotherapy (MD −19.9 [−29.0; −10.7] to −11.5 [−17.5; −5.6], p < 0.001) and stem cell transplantation (HR-SCT) (MD −24.7 [−30.1; −19.4] to −16.9 [−27.4; −6.5], p < 0.001 to p = 0.002) as well as self-reports from HR (MD −11.2 [−18.9; −3.4], p = 0.006) and HR-SCT survivors (MD −14.5 [−24.1; −4.8], p = 0.004). Among 5–15-year-olds, PFD/ + BDR was associated with significantly poorer parent proxy-reported physical functioning in survivors versus controls (MD −14.3 [−21.2; −7.3], p < 0.001). Significant deficits in these groups related to running, engaging in sports/exercise and lifting heavy objects. PFD/ + BDR was not associated with poorer physical functioning reports within survivors (MD −2.5 [−9.0; 3.3] to 0.1 [−13.7; 13.8], p = 0.36–0.99).

Conclusions
Physical functioning in survivors compared to controls varies with intensity of treatment, most evident in HR and HR-SCT survivors, with impairments linked to PFD and +BDR in the youngest. Among survivors, physical functioning did not align with PFD and +BDR status, highlighting the need for parallel assessment of objective and subjective measures during follow-up. Baseline PFT and tailored follow-up are essential for high-risk groups, to detect PFD early and intervene.
OriginalsprogEngelsk
Artikelnummere71389
TidsskriftPediatric Pulmonology
Vol/bind60
Udgave nummer11
Antal sider15
ISSN1054-187X
DOI
StatusUdgivet - 2025

Bibliografisk note

© 2025 The Author(s). Pediatric Pulmonology published by Wiley Periodicals LLC.

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