Research output per year
Research output per year
Nørre Allé 20
2200 København N
Research activity per year
Martin Nilsson
Vismarlövsvägen 179-22
233 76 Klågerup
Tel: 46 70 2373566 (mobile),
690514-4231, 140569-3239
Personal
Born May 14th 1969 in Lund, Sweden. Swedish citizen. Married, two children
Professional experience
1501- Copenhagen University, Assistant Professor
0811-1412 Copenhagen University, Postdoctoral Position
0802-0810 DTU, Postdoctoral Position
0509-0708 Affibody AB, Scientist
0501-0508 Affibody AB, Head of Affibody Generation
0311-0412 Affibody AB, Team leader
0105-0310 Biostapro/Department of Microbiology, SLU, Researcher
9501-0104 Department of Microbiology, SLU, PhD student
9406-9408 Department of Molecular biology, Uppsala University
9305-9312 Department of Microbiology, SLU
Education
95-01 PhD student at the Department of Microbiology at SLU
89-94 Master of Science in Agriculture at the Swedish University of Agricultural Sciences (Biotechnology program)
85-88 Nature science, Berga, Eslöv
Publications
23 peer-reviewed articles
Doctoral theses. 2001. Fibrinogen and von Willebrand factor binding proteins in Staphylococci
New fibrinogen binding protein originating from coagulase-negative Staphylococcus. 1997. Applicants and Inventors: Guss, Bengt; Nilsson, Martin; Frykberg, Lars; Flock, Jan-Ingmar; Lindberg, Martin (EPO 97928617.6)
Von Willebrand binding factor-binding proteins from staphylococci. 2000. Guss, Bengt; Nilsson, Martin; Frykberg, Lars; Jacobsson Karin; Bjerketorp Joakim (PCT/SE017/00766)
Education
Master of Science in Biotechnology from SLU, Uppsala
PhD in Microbiology from SLU, Uppsala
Previous employment
Affibody AB, Scientist, Team leader, 2003-2007
DTU and Copenhagen University, Postdoctoral Position 2008-2014
At Costerton Biofilm Center I work with various biofilm related projects. A transposon library based on the oral pathogen Streptococcus mutans was used to identify genes involved in antimicrobial tolerance in biofilms. Thereafter, the tolerance mechanisms mediated by the products of the isolated genes were investigated. Currently, I am part of a team that focus on identifying and studying compounds that interfere with biofilm formation in Pseudomonas aeruginosa. In addition, I have also contributed in other studies e.g. the function of amyloids in biofilm formation in Pseudomonas species, the presence of biofilms in skin biopsis from patients with Hidradenitis suppurativa and if DNA from oral bacteria can be detected on coronary angioplasty balloons collected from patients.
Research output: Contribution to journal › Journal article › Research › peer-review
Research output: Contribution to journal › Journal article › Research › peer-review
Research output: Contribution to journal › Journal article › Research › peer-review
Research output: Contribution to journal › Journal article › Research › peer-review
Research output: Contribution to journal › Journal article › Research › peer-review
Research output: Contribution to journal › Journal article › Research › peer-review
Research output: Contribution to journal › Journal article › Research › peer-review
Research output: Contribution to journal › Journal article › Research › peer-review