No photo of Carlos Henriquez Olguín
  • Source: Scopus
20092024

Research activity per year

Personal profile

Short presentation

The rapid rise in the prevalence of obesity, diabetes, and physical inactivity has significantly contributed to the increasing global burden of noncommunicable diseases. Skeletal muscle, the largest tissue in the body, possesses remarkable plasticity and represents a key pharmacological target to combat these diseases. Skeletal muscle redox signaling has emerged as a novel regulator of skeletal muscle metabolism but the molecular mechanisms involved are still not well understood. Our research focuses on connecting intracellular redox signaling and skeletal muscle metabolism in the context of metabolic diseases.  

 

Recent Selected Publications 

Henriquez-Olguin, C., Knudsen, J.R., Raun, S.H. et al. Cytosolic ROS production by NADPH oxidase 2 regulates muscle glucose uptake during exercise. Nature Commun 10, 4623 (2019).

Henriquez-Olguin C., Boronat, S.  et al.  NADPH oxidases in skeletal muscle: Emerging roles in redox signaling and metabolism. Antioxid Redox Signal. 31(18):1371-1410 (2019)

Henriquez-Olguin C, Baghersad L, Arab-Ceschia L, Raun SH., et al.  Adaptations to high-intensity interval training in skeletal muscle require NADPH oxidase 2. Redox Biology 24, 101188 (2019)

Raun, S.H., Henriquez-Olguin, C., Karavaeva, I. et al. Housing temperature influences exercise training adaptations in mice. Nature Commun 11, 1560 (2020).

 

Primary fields of research

- Skeletal Muscle Metabolism 

- Redox Signaling 

- Insulin Action 

- Exercise Physiology 

 

ResearcherID: K-7671-2012

Scopus Author ID: 35102305800

Education/Academic qualification

PhD in Molecular Physiology , University of Copenhagen

Award Date: 30 Sep 2018

PhD in Biomedical Sciences , University of Chile

Award Date: 6 Aug 2018

Master in Physiology , University of Chile

Award Date: 17 Jun 2013

Keywords

  • Faculty of Science
  • Insulin sensitivity
  • Skeletal muscle
  • Redox signaling
  • Reactive oxygen species
  • Exercise physiology
  • Insulin resistance

Collaborations and top research areas from the last five years

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