Increased hepatic glycogen synthetase and decreased phosphorylase in trained rats

H Galbo; P Saugmann; Erik A. Richter

doi:10.1111/j.1748-1716.1979.tb06473.x

Increased hepatic glycogen synthetase and decreased phosphorylase in trained rats

H Galbo, P Saugmann, Erik A. Richter

Research output: Contribution to journal › Journal article › Research › peer-review

16 Citations (Scopus)

Abstract

Rats were either physically trained by a 12 wk swimming program or were freely eating or weight matched, sedentary controls. Trained rats had a higher relative liver weight and total hepatic glycogen synthetase (EC 2.4.1.11) activity and a lower phosphorylase (EC 2.4.1.1) activity than the other groups of rats. These changes may partly explain the demonstrated training-induced increase in glucose tolerance. None of the findings could be ascribed to differences in foold intake or body weight.

Original language	English
Journal	Acta Physiologica Scandinavica
Volume	107
Issue number	3
Pages (from-to)	269-272
Number of pages	4
ISSN	0001-6772
DOIs	https://doi.org/10.1111/j.1748-1716.1979.tb06473.x
Publication status	Published - 1979

Keywords

Animals
Blood Glucose
Body Weight
Glucose Tolerance Test
Glycogen Synthase
Liver
Liver Glycogen
Male
Organ Size
Phosphorylases
Physical Conditioning, Animal
Rats

Access to Document

10.1111/j.1748-1716.1979.tb06473.x

Cite this

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title = "Increased hepatic glycogen synthetase and decreased phosphorylase in trained rats",

abstract = "Rats were either physically trained by a 12 wk swimming program or were freely eating or weight matched, sedentary controls. Trained rats had a higher relative liver weight and total hepatic glycogen synthetase (EC 2.4.1.11) activity and a lower phosphorylase (EC 2.4.1.1) activity than the other groups of rats. These changes may partly explain the demonstrated training-induced increase in glucose tolerance. None of the findings could be ascribed to differences in foold intake or body weight.",

keywords = "Animals, Blood Glucose, Body Weight, Glucose Tolerance Test, Glycogen Synthase, Liver, Liver Glycogen, Male, Organ Size, Phosphorylases, Physical Conditioning, Animal, Rats",

author = "H Galbo and P Saugmann and Richter, {Erik A.}",

year = "1979",

doi = "10.1111/j.1748-1716.1979.tb06473.x",

language = "English",

volume = "107",

pages = "269--272",

journal = "Acta Physiologica Scandinavica",

issn = "0001-6772",

publisher = "Blackwell Science Ltd.",

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TY - JOUR

T1 - Increased hepatic glycogen synthetase and decreased phosphorylase in trained rats

AU - Galbo, H

AU - Saugmann, P

AU - Richter, Erik A.

PY - 1979

Y1 - 1979

N2 - Rats were either physically trained by a 12 wk swimming program or were freely eating or weight matched, sedentary controls. Trained rats had a higher relative liver weight and total hepatic glycogen synthetase (EC 2.4.1.11) activity and a lower phosphorylase (EC 2.4.1.1) activity than the other groups of rats. These changes may partly explain the demonstrated training-induced increase in glucose tolerance. None of the findings could be ascribed to differences in foold intake or body weight.

AB - Rats were either physically trained by a 12 wk swimming program or were freely eating or weight matched, sedentary controls. Trained rats had a higher relative liver weight and total hepatic glycogen synthetase (EC 2.4.1.11) activity and a lower phosphorylase (EC 2.4.1.1) activity than the other groups of rats. These changes may partly explain the demonstrated training-induced increase in glucose tolerance. None of the findings could be ascribed to differences in foold intake or body weight.

KW - Animals

KW - Blood Glucose

KW - Body Weight

KW - Glucose Tolerance Test

KW - Glycogen Synthase

KW - Liver

KW - Liver Glycogen

KW - Male

KW - Organ Size

KW - Phosphorylases

KW - Physical Conditioning, Animal

KW - Rats

U2 - 10.1111/j.1748-1716.1979.tb06473.x

DO - 10.1111/j.1748-1716.1979.tb06473.x

M3 - Journal article

C2 - 120110

VL - 107

SP - 269

EP - 272

JO - Acta Physiologica Scandinavica

JF - Acta Physiologica Scandinavica

SN - 0001-6772

IS - 3

ER -